By Dr. Jan A. L. Bulcke, Professor Dr. Albert L. Baert (auth.)
One of the main difficult and outstanding good points of a number of the genetically made up our minds revolutionary neuromuscular ailments resembling the spinal muscular atrophies and the muscular dystrophies is that muscular losing and vulnerable ness in those instances is interestingly selective, a minimum of within the early levels, decide ing out sure skeletal muscular tissues and sparing others. The analysis of those stipulations has mostly depended long ago upon the popularity of particular styles of involvement of person muscle tissue and muscle groups, taken besides info derived from the mode of inheritance in the in dividual relations and the result of distinctive investigations. The investigations of such a lot worth have proved to be serum enzyme reviews, electromyography and comparable suggestions, and muscle biopsy. the arrival of CT scanning has, although, brought a brand new measurement; because the authors of this attention-grabbing monograph have sincerely proven, it really is now attainable, utilizing the full physique scanner, to outline styles of muscular atrophy within the limbs and trunk even more accurately than by means of the other process. not just does this techni que show which muscle groups are concerned, however the alterations in relative density supply beneficial information regarding the severity of the method and concerning the development of the illness if the experiences are played serially. This monograph is pleasantly written and so much attractively illustrated.
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Additional info for Clinical and Radiological Aspects of Myopathies: CT Scanning · EMG · Radioisotopes
Ambiguus nu. ambiguus nu. ambiguus nu. ambiguus nu. ambiguus nu. ambiguus nu. ambiguus Motor nu. n. facialis Motor nu. n. trigemini nu. ambiguus Motor nu. n. /J 50" ..... C/O t: n t: ~ E. /J ::s ~ S ::r: t: (1) :T 0 -, ::s o· ;:? < 0' ..... (1) 0t: ..... (1) n '"0 ..... 0 > tv V1 m. constrictor pharyngis medius m. constrictor pharyngis inferior Longitudinal muscles m.
In the third and most common type all signs are essentially neurogenic. As the cerebral atrophy progresses spasticity also appears. In the fourth type the muscles may initially be hypotrophic, but without either neurogenic or myogenic features. The fifth, and most controversial type shows signs of an arrested, burned out myopathy. Some of these myopathic forms of AMC have been described as separate entities such als Ullrich's disease. NIHEI et al. (1979) described 15 cases of this syndrome, which consists of proximal joint contractures, muscular hypotonia, prominent calcaneus, high arched palate and normal intelligence.
Dividual muscles, selective atrophy of specific muscles and muscle groups and asymmetry between two identical but contralateral muscles. No explanation for this phenomenon could be given. Other studies by the same author have described soft tissue X-ray findings in muscular pseudohypertrophy (PALVOLGYI and GALLAI 1979), anterior horn cell lesions (PALVOLGYI 1979), closed muscle and tendon injuries of the upper arm (PALVOLGYI and BALINT 1979) and lacerations in tendons and muscles in the Ehlers-Danlos syndrome (PALVOLGYI et al.